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Publication

LP342, a novel histone deacetylase inhibitor, decreases nitro-oxidative stress, mitochondrial dysfunction and hepatic ischaemia/reperfusion injury in mice.

Devadoss J Samuvel | Yasodha Krishnasamy | Li Li | John J Lemasters | C James Chou | Zhi Zhong
RPS pharmacy and pharmacology reports | 2023

Some histone deacetylase (HDAC) isoforms contribute to ischaemia/reperfusion (IR) injury (IRI). Here, we examined whether LP342, the lead candidate of a new generation of hydrazide-based HDAC inhibitors (HDACi), decreases hepatic IRI.

Pubmed ID: 37092117

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Associated grants

  • Agency: NIH HHS, United States
    Id: S10 OD018113
  • Agency: NIDDK NIH HHS, United States
    Id: P30 DK123704
  • Agency: NIDDK NIH HHS, United States
    Id: R41 DK130707
  • Agency: NIDDK NIH HHS, United States
    Id: R41 DK120247
  • Agency: NIGMS NIH HHS, United States
    Id: P20 GM130457
  • Agency: NIDDK NIH HHS, United States
    Id: R01 DK119523
  • Agency: NIAAA NIH HHS, United States
    Id: R01 AA025379
  • Agency: NIGMS NIH HHS, United States
    Id: P30 GM140964
  • Agency: NCI NIH HHS, United States
    Id: P30 CA138313
  • Agency: NIGMS NIH HHS, United States
    Id: P20 GM103542

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C57BL/6J (tool)

RRID:IMSR_JAX:000664

Mus musculus with name C57BL/6J from IMSR.

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The Stimulating Peripheral Activity to Relieve Conditions (SPARC) effort is the result of National Institutes of Health’s (NIH) drive to map out the neural circuitry responsible for visceral control in higher vertebrates. The SPARC Anatomy Working Group (SAWG) is responsible for the integrity of anatomical knowledge in SPARC

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