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Publication

High Residual β-cell Function in Chinese Patients With Autoimmune Type 1 Diabetes.

Yueshu Wang | Yao Qin | Huilan Gu | Linyu Zhang | Jing Wang | Yiting Huang | Yuwen Shi | Qizhen Hu | Yang Chen | Yong Gu | Yun Shi | Yang Tao | Mei Zhang
The Journal of clinical endocrinology and metabolism | 2022

The destruction of pancreatic β cells causes type 1 diabetes mellitus (T1D), an autoimmune disease. Studies have demonstrated that there is heterogeneity in residual β-cell function in Caucasians; therefore, we aimed to evaluate β-cell function in Chinese autoimmune T1D patients.

Pubmed ID: 35218654

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Publication data is provided by the National Library of Medicine ® and PubMed ®. Data is retrieved from PubMed ® on a weekly schedule. For terms and conditions see the National Library of Medicine Terms and Conditions.

This is a list of tools and resources that we have found mentioned in this publication.

Isletest™ - islet cell autoantigen (IAA) ELISA (antibody)

RRID:AB_2895270

This unknown targets

View all literature mentions

pancreas islets Kit (antibody)

RRID:AB_2895268

This unknown targets

View all literature mentions

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The Stimulating Peripheral Activity to Relieve Conditions (SPARC) effort is the result of National Institutes of Health’s (NIH) drive to map out the neural circuitry responsible for visceral control in higher vertebrates. The SPARC Anatomy Working Group (SAWG) is responsible for the integrity of anatomical knowledge in SPARC

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