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Publication

Chidamide inhibits t(8;21) AML cell proliferation and AMK1/ETO and C-KIT expression by inhibiting ERK1/2 signaling pathway.

Jing Liu | Na Lv | Lei Zhou | Yan Li | Li Yu
Translational cancer research | 2020

t(8;21) acute myeloid leukemia (AML) is a highly heterogenous hematological malignancy. Histone deacetylases inhibitors (HDACi) are a group of small-molecule compounds with extensive anti-tumor activity. Chidamide (CS055) is a selective HDACi independently developed by China. We aimed to investigate its anti-tumor activity and mechanism in t(8;21) AML cells.

Pubmed ID: 35117428

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U-937 (tool)

RRID:CVCL_0007

Cell line U-937 is a Cancer cell line with a species of origin Homo sapiens (Human)

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Kasumi-1 (tool)

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Cell line Kasumi-1 is a Cancer cell line with a species of origin Homo sapiens (Human)

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HL-60 (tool)

RRID:CVCL_0002

Cell line HL-60 is a Cancer cell line with a species of origin Homo sapiens (Human)

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