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Publication

Association of Epstein-Barr virus serological reactivation with transitioning to systemic lupus erythematosus in at-risk individuals.

Neelakshi R Jog | Kendra A Young | Melissa E Munroe | Michael T Harmon | Joel M Guthridge | Jennifer A Kelly | Diane L Kamen | Gary S Gilkeson | Michael H Weisman | David R Karp | Patrick M Gaffney | John B Harley | Daniel J Wallace | Jill M Norris | Judith A James
Annals of the rheumatic diseases | 2019

Systemic lupus erythematosus (SLE) is a systemic autoimmune disease with unknown aetiology. Epstein-Barr virus (EBV) is an environmental factor associated with SLE. EBV maintains latency in B cells with frequent reactivation measured by antibodies against viral capsid antigen (VCA) and early antigen (EA). In this study, we determined whether EBV reactivation and single nucleotide polymorphisms (SNPs) in EBV-associated host genes are associated with SLE transition.

Pubmed ID: 31217170

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Associated grants

  • Agency: NIGMS NIH HHS, United States
    Id: U54 GM104938
  • Agency: NIAID NIH HHS, United States
    Id: U19 AI082714
  • Agency: NIAMS NIH HHS, United States
    Id: P30 AR053483
  • Agency: NIAMS NIH HHS, United States
    Id: P30 AR073750
  • Agency: NIAID NIH HHS, United States
    Id: U01 AI101934

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HEp-2 (tool)

RRID:CVCL_1906

Cell line HEp-2 is a Cancer cell line with a species of origin Homo sapiens (Human)

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The Stimulating Peripheral Activity to Relieve Conditions (SPARC) effort is the result of National Institutes of Health’s (NIH) drive to map out the neural circuitry responsible for visceral control in higher vertebrates. The SPARC Anatomy Working Group (SAWG) is responsible for the integrity of anatomical knowledge in SPARC

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