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Publication

Bone Marrow-Derived Endothelial Progenitor Cells Contribute to Monocrotaline-Induced Pulmonary Arterial Hypertension in Rats via Inhibition of Store-Operated Ca2+ Channels.

Ran Miao | Jun Wan | Jie Liu | Jason X-J Yuan | Jing Wang | Wanmu Xie | Zhenguo Zhai | Chen Wang
BioMed research international | 2018

This study aimed to explore whether bone marrow- (BM-) derived endothelial progenitor cells (EPCs) contributing to monocrotaline- (MCT-) induced pulmonary arterial hypertension (PAH) in rats via modulating store-operated Ca2+ channels (SOC).

Pubmed ID: 30320134

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Associated grants

  • Agency: NIEHS NIH HHS, United States
    Id: P30 ES006694
  • Agency: NHLBI NIH HHS, United States
    Id: R35 HL135807
  • Agency: NHLBI NIH HHS, United States
    Id: U01 HL125208

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SD (tool)

RRID:RGD_70508

Rattus norvegicus with name SD from RGD.

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The Stimulating Peripheral Activity to Relieve Conditions (SPARC) effort is the result of National Institutes of Health’s (NIH) drive to map out the neural circuitry responsible for visceral control in higher vertebrates. The SPARC Anatomy Working Group (SAWG) is responsible for the integrity of anatomical knowledge in SPARC

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