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Publication

Autophagy gene ATG5 knockdown upregulates apoptotic cell death during Candida albicans infection in human vaginal epithelial cells.

Ankit Shroff | Kudumula Venkata Rami Reddy
American journal of reproductive immunology (New York, N.Y. : 1989) | 2018

Candida albicans infection induces damage in host cells that results in the activation of cell death pathways such as apoptosis and necrosis. Autophagy acts as a pro-survival mechanism during various infections and has cross talks with apoptosis. The objective of our study was to delineate the effect of autophagy knockdown in human vaginal epithelial cells (VECs) during Candida infection-induced apoptosis.

Pubmed ID: 30303264

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Associated grants

  • Agency: Department of Biotechnology (DBT), International
  • Agency: Indian Council of Medical Research (ICMR), International
  • Agency: Govt. of India, International
  • Agency: NIRRH, International
    Id: NIRRH/RA/663/2018

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The Stimulating Peripheral Activity to Relieve Conditions (SPARC) effort is the result of National Institutes of Health’s (NIH) drive to map out the neural circuitry responsible for visceral control in higher vertebrates. The SPARC Anatomy Working Group (SAWG) is responsible for the integrity of anatomical knowledge in SPARC

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