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Publication

Intracellular rupture, exocytosis and actin interaction of endocytic vacuoles in pancreatic acinar cells: initiating events in acute pancreatitis.

Michael Chvanov | Francesca De Faveri | Danielle Moore | Mark W Sherwood | Muhammad Awais | Svetlana Voronina | Robert Sutton | David N Criddle | Lee Haynes | Alexei V Tepikin
The Journal of physiology | 2018

Giant trypsin-containing endocytic vacuoles are formed in pancreatic acinar cells stimulated with inducers of acute pancreatitis. F-actin envelops endocytic vacuoles and regulates their properties. Endocytic vacuoles can rupture and release their content into the cytosol of acinar cells. Endocytic vacuoles can fuse with the plasma membrane of acinar cells and exocytose their content.

Pubmed ID: 29717784

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Associated grants

  • Agency: Department of Health, United Kingdom
    Id: EME/15/20/01
  • Agency: Medical Research Council, United Kingdom
    Id: MR/K012967/1
  • Agency: Wellcome Trust, United Kingdom
    Id: 105273/Z/14/A

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RRID:SCR_008452

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The Stimulating Peripheral Activity to Relieve Conditions (SPARC) effort is the result of National Institutes of Health’s (NIH) drive to map out the neural circuitry responsible for visceral control in higher vertebrates. The SPARC Anatomy Working Group (SAWG) is responsible for the integrity of anatomical knowledge in SPARC

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