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Publication

Nitric oxide and protein kinase G act on TRPC1 to inhibit 11,12-EET-induced vascular relaxation.

Peng Zhang | Yan Ma | Yan Wang | Xin Ma | Yu Huang | Ronald A Li | Song Wan | Xiaoqiang Yao
Cardiovascular research | 2014

Vascular endothelial cells synthesize and release vasodilators such as nitric oxide (NO) and epoxyeicosatrienoic acids (EETs). NO is known to inhibit EET-induced smooth muscle hyperpolarization and relaxation. This study investigates the underlying mechanism of this inhibition.

Pubmed ID: 25139746

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TRPC1-FLAG (tool)

RRID:Addgene_24408

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The Stimulating Peripheral Activity to Relieve Conditions (SPARC) effort is the result of National Institutes of Health’s (NIH) drive to map out the neural circuitry responsible for visceral control in higher vertebrates. The SPARC Anatomy Working Group (SAWG) is responsible for the integrity of anatomical knowledge in SPARC

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