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Publication

Long term liver specific glucokinase gene defect induced diabetic cardiomyopathy by up regulating NADPH oxidase and down regulating insulin receptor and p-AMPK.

Hui Li | Xi Wang | Yiqing Mao | Ruobi Hu | Wei Xu | Zhen Lei | Na Zhou | Ling Jin | Tingting Guo | Zhixin Li | David M Irwin | Gang Niu | Huanran Tan
Cardiovascular diabetology | 2014

The liver-specific glucokinase knockout (gckw/-) mouse experiences long-term hyperglycemia and insulin resistance. This study was designed to evaluate the functional and structural changes in the myocardium of 60 week-old gckw/- mice, and to investigate the effect of rosiglitazone on the myocardium in this model.

Pubmed ID: 24447392

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The Stimulating Peripheral Activity to Relieve Conditions (SPARC) effort is the result of National Institutes of Health’s (NIH) drive to map out the neural circuitry responsible for visceral control in higher vertebrates. The SPARC Anatomy Working Group (SAWG) is responsible for the integrity of anatomical knowledge in SPARC

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