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Publication

Vascular endothelial growth factor regulates myeloid cell leukemia-1 expression through neuropilin-1-dependent activation of c-MET signaling in human prostate cancer cells.

Shumin Zhang | Haiyen E Zhau | Adeboye O Osunkoya | Shareen Iqbal | Xiaojian Yang | Songqing Fan | Zhengjia Chen | Ruoxiang Wang | Fray F Marshall | Leland W K Chung | Daqing Wu
Molecular cancer | 2010

Myeloid cell leukemia-1 (Mcl-1) is a member of the Bcl-2 family, which inhibits cell apoptosis by sequestering pro-apoptotic proteins Bim and Bid. Mcl-1 overexpression has been associated with progression in leukemia and some solid tumors including prostate cancer (PCa). However, the regulatory mechanism for Mcl-1 expression in PCa cells remains elusive.

Pubmed ID: 20085644

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Associated grants

  • Agency: NCI NIH HHS, United States
    Id: R01 CA122602
  • Agency: NCI NIH HHS, United States
    Id: R01 CA256058
  • Agency: NCI NIH HHS, United States
    Id: 1R43CA141870
  • Agency: NCI NIH HHS, United States
    Id: P01 CA98912

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HUVEC-C (tool)

RRID:CVCL_2959

Cell line HUVEC-C is a Finite cell line with a species of origin Homo sapiens

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The Stimulating Peripheral Activity to Relieve Conditions (SPARC) effort is the result of National Institutes of Health’s (NIH) drive to map out the neural circuitry responsible for visceral control in higher vertebrates. The SPARC Anatomy Working Group (SAWG) is responsible for the integrity of anatomical knowledge in SPARC

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