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Publication

KDELR2 knockdown synergizes with temozolomide to induce glioma cell apoptosis through the CHOP and JNK/p38 pathways.

Guofeng Zhang | Bin Wang | Shiqi Cheng | Hengyi Fan | Shaowen Liu | Bin Zhou | Weibin Liu | Rui Liang | Youjia Tang | Yan Zhang
Translational cancer research | 2021

The C-terminal tetrapeptide Lys-Asp-Glu-Leu receptors (KDELRs) are transmembrane proteins that regulate ER stress (ERS) response, growth, differentiation, and immune responses. There is an association between KDELR2and promotion of glioblastoma tumorigenesis. The aim of the present study was to explore the functional mechanism of KDELR2 in glioma and during response to chemotherapy to temozolomide (TMZ).

Pubmed ID: 35116653

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