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Neutrophil depletion reduces edema formation and tissue loss following traumatic brain injury in mice.

Ellinor Kenne | Anna Erlandsson | Lennart Lindbom | Lars Hillered | Fredrik Clausen
Journal of neuroinflammation | 2012

Brain edema as a result of secondary injury following traumatic brain injury (TBI) is a major clinical concern. Neutrophils are known to cause increased vascular permeability leading to edema formation in peripheral tissue, but their role in the pathology following TBI remains unclear.

Pubmed ID: 22269349

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Bio X Cell (tool)

RRID:SCR_004997

Commercial supplier and developer of in vivo antibodies. Provides antibodies and antibody production services.

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C57BL/6J (tool)

RRID:IMSR_JAX:000664

Mus musculus with name C57BL/6J from IMSR.

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