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Publication

BPR1J-097, a novel FLT3 kinase inhibitor, exerts potent inhibitory activity against AML.

W-H Lin | W-T Jiaang | C-W Chen | K-J Yen | S-Y Hsieh | S-C Yen | C-P Chen | K-Y Chang | C-Y Chang | T-Y Chang | Y-L Huang | T-K Yeh | Y-S Chao | C-T Chen | J T-A Hsu
British journal of cancer | 2012

Activating mutations of Fms-like tyrosine kinase 3 (FLT3) constitute a major driver in the pathogenesis of acute myeloid leukaemia (AML). Hence, pharmacological inhibitors of FLT3 are of therapeutic interest for AML.

Pubmed ID: 22187040

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