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Stimulating the central nervous system to prevent intestinal dysfunction after traumatic brain injury.

V Bansal | T Costantini | SY Ryu | C Peterson | W Loomis | J Putnam | B Elicieri | A Baird | R Coimbra
The Journal of trauma | 2010 May

Traumatic brain injury (TBI) causes gastrointestinal dysfunction and increased intestinal permeability. Regulation of the gut barrier may involve the central nervous system. We hypothesize that vagal nerve stimulation prevents an increase in intestinal permeability after TBI.

Pubmed ID: 20453760

TBI Model

  • Weight-drop model
  • Weight-drop

    • Animal Information

  • Species: mouse
  • Strain: balb/c
  • Age (weeks): No age reported
  • Weight (grams): 20 - 24

    • Assessments

    Histologic, In Vivo Intestinal Permeability Assay, Levels of Intestinal TNF-α, Polymerase Chain Reaction of Intestinal Glial Fibrillary Acidic Protein

    TBI model parameters

  • Impact Depth (mm): No information available
  • Impact Duration (ms): No information available
  • Impact Velocity (m/s): No information available
  • Impactor Tip: No information available

    • Device Name

    No information available

    Associated Datasets

    No information available

    Associated Protocols

    No information available

    About

    The PRECISE-TBI toolbox are a collection tools and resources to increase rigor and reproducibility with Traumatic Brain Injury within the PRE Clinical Interagency reSearch resourcE - Traumatic Brain Injury (PRECISE-TBI). Please learn more about PRECISE-TBI at https://www.precise-tbi.org

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    Contact Us

    Department of Neurosciences, School of Medicine

    University of California, San Diego





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