Searching across hundreds of databases

Our searching services are busy right now. Your search will reload in five seconds.

  • Register
X
Forgot Password

If you have forgotten your password you can enter your email here and get a temporary password sent to your email.

X

Leaving Community

Are you sure you want to leave this community? Leaving the community will revoke any permissions you have been granted in this community.

No
Yes
X
Forgot Password

If you have forgotten your password you can enter your email here and get a temporary password sent to your email.

Search

Type in a keyword to search

on page 2 showing 24 out of 3,318,841 results
https://www.addgene.org/101270/

Cite This: RRID:Addgene_101270
5WCJ; gi|55956896
Species: Homo sapiens
Genetic insert: METTL13
Vector Backbone: pFBOH-MHL
Reference:
Comment: N terminal tag: MHHHHHHSSGRENLYFQG. SGC Clone Sample ID: METTL13:PBC007-B09:C235684. SGC or PDB link: http://www.thesgc.org/structures/5WCJ/


Cite this (MMRRC Cat# 041197-JAX, RRID:MMRRC_041197-JAX)

Source Database: MMRRC, catalog # 041197-JAX
Genetic Background: Spontaneous Mutation
Affected Genes: Dmd
Genomic Alteration:
Availability:
Reference:
Notes: abnormal skeletal muscle fiber morphology [MP:0003084], centrally nucleated skeletal muscle fibers [MP:0009404], skeletal muscle fiber necrosis [MP:0009414], myocardial necrosis [MP:0006085], homeostasis/metabolism phenotype [MP:0005376], increased circulating creatine kinase level [MP:0010090], increased growth hormone level [MP:0005137], abnormal corticotroph morphology [MP:0008327], increased somatotroph cell size [MP:0008416], impaired coordination [MP:0001405], abnormal grip strength [MP:0001515], abnormal auditory brainstem response waveform shape [MP:0011966], increased or absent threshold for auditory brainstem response [MP:0011967], increased susceptibility to noise-induced hearing loss [MP:0004597], increased interferon-gamma secretion [MP:0008566], increased interleukin-12 secretion [MP:0008663], increased interleukin-2 secretion [MP:0008687], increased interleukin-4 secretion [MP:0008699], increased interleukin-6 secretion [MP:0008705], increased tumor necrosis factor secretion [MP:0008560], abnormal muscle morphology [MP:0002108], dilated sarcoplasmic reticulum [MP:0004089], increased variability of skeletal muscle fiber size [MP:0009403], skeletal muscle fibrosis [MP:0009419], skeletal muscle necrosis [MP:0003852], dystrophic muscle [MP:0000752], muscle degeneration [MP:0000749], muscular atrophy [MP:0002269], abnormal muscle physiology [MP:0002106], myopathy [MP:0000751], abnormal circulating pyruvate kinase level [MP:0010106], reduced female fertility [MP:0001923], abnormal skeletal muscle morphology [MP:0000759], increased skeletal muscle fiber diameter [MP:0009401], abnormal diaphragm morphology [MP:0002279], abnormal intercostal muscle morphology [MP:0002280], increased skeletal muscle mass [MP:0004818], skeletal muscle hypertrophy [MP:0009461], skeletal muscle endomysial fibrosis [MP:0009420], abnormal skeletal muscle satellite cell proliferation [MP:0009410], abnormal muscle contractility [MP:0005620], abnormal muscle electrophysiology [MP:0004145], abnormal muscle tone [MP:0004142], decreased physiological sensitivity to xenobiotic [MP:0008874], nuclear cataracts [MP:0010254], anterior subcapsular cataracts [MP:0010252], nervous system phenotype [MP:0003631], abnormal neuromuscular synapse morphology [MP:0001053], impaired exercise endurance [MP:0012106], increased transforming growth factor level [MP:0008837], skeletal muscle fiber degeneration [MP:0009412], impaired skeletal muscle contractility [MP:0002841], abnormal vertebral column morphology [MP:0004703] Research areas: Cardiovascular, Cell Biology, Developmental Biology, Models for Human Disease; Mutation type: Spontaneous Mutation


https://web.expasy.org/cellosaurus/CVCL_8X21

Cite this: (ECACC Cat# 89042138, RRID:CVCL_8X21)
Organism: Homo sapiens
Disease:
Category: Finite cell line DT Created: 23-02-16; Last updated: 07-09-18; Version: 2
Comment: Part of: ECACC chromosomal abnormality collection. DT Created: 23-02-16; Last updated: 07-09-18; Version: 2


https://www.ncbi.nlm.nih.gov/biosample/SAMN08619388

Cite this: (IIDP, Cat# 1986, RRID:SAMN08619388)
Organism: Homo sapiens
Disease: Diabetes
Category: Tissue: Pancreas
Comment: IIDP Submission for Human Islets; Provider: Camilo Ricordi,University of Miami, Miami, FL, 33136; Age: 60.00 Years; Submission Date: 2018-02-27T22:50:04.596; Status of tissue: live


http://antibodyregistry.org/AB_614357

Cite this (IMGENEX Cat# IMG-4128, RRID:AB_614357)
Comments: manufacturer recommendations: IgG; IgG WB; Western Blot
References:
Host Organism: rabbit
Clonality: polyclonal antibody
Target(s): XAB2/HCNP


Cite this (Time Event Ontology, RRID:SCR_000310)

URL: http://purl.bioontology.org/ontology/TEO

Resource Type: Resource, ontology, data or information resource, controlled vocabulary

Ontology for representing events, time, and their relationships.


https://www.addgene.org/60817/

Cite This: RRID:Addgene_60817
binding site of bacteriophage λ antitermination protein N; boxb
Species: Synthetic
Genetic insert: 25x BoxB
Vector Backbone: pCMV5
Reference: PMID:24035393
Comment: the authors of Gehring et al. Mol Cell. 2003 Apr;11(4):939-49 were contacted by us and granted their permission to distribute the 25xboxb construct generated in our lab from their precursor for non-profit purpose via Addgene


Cite this (MGI Cat# 3717918, RRID:MGI:3717918)

Source Database: MGI, catalog # 3717918
Genetic Background: BKa.129-Sox17
Affected Genes: Sox17
Genomic Alteration: tm1Sjm
Availability: Availability unknown check source stock center
Reference: PMID:17655922
Notes: liver hypoplasia, embryonic lethality during organogenesis, complete penetrance, decreased hematopoietic stem cell number, abnormal embryonic hematopoiesis, abnormal embryo turning, abnormal rostral-caudal axis patterning, embryonic growth retardation Allele Detail: Targeted


https://web.expasy.org/cellosaurus/CVCL_8V84

Cite this: (ECACC Cat# 91053109, RRID:CVCL_8V84)
Organism: Homo sapiens
Disease:
Category: Transformed cell line DT Created: 23-02-16; Last updated: 07-09-18; Version: 3
Comment: Part of: ECACC chromosomal abnormality collection. Transformant: NCBI_TaxID; 10376; Epstein-Barr virus (EBV). DT Created: 23-02-16; Last updated: 07-09-18; Version: 3


https://www.ncbi.nlm.nih.gov/biosample/SAMN08617638

Cite this: (IIDP, Cat# 1982, RRID:SAMN08617638)
Organism: Homo sapiens
Disease: Diabetes
Category: Tissue: Pancreas
Comment: IIDP Submission for Human Islets; Provider: Fouad Kandeel,Southern California Islet Cell Resources Center, Duarte, CA, 91010; Age: 49.00 Years; Submission Date: 2018-02-27T14:56:04.543; Status of tissue: live


http://antibodyregistry.org/AB_2267743

Cite this (R and D Systems Cat# MAB2627, RRID:AB_2267743)
Comments: vendor recommendations: IgG1 Immunoprecipitation; Western Blot; Immunoprecipitation, Western Blot
References:
Host Organism: rat
Clonality: monoclonal antibody
Target(s): Human PCPE-1 MAb (Clone 261730)


Cite this (Institute for Advanced Biosciences, RRID:SCR_008526)

URL: http://g2d2.ogic.ca

Resource Type: Resource, organization portal, data or information resource, portal, database

The Institute for Advanced Biosciences, Keio University, is an academic research institute pioneering the new life science field of Systems Biology, using both experimental and computational biology. There are several groups working in collaboration, focusing mainly on genome biology and engineering, genome design and synthetic biology, metabolic engineering, proteomics, metabolomics, RNA biology, bioinformatics and computational biology. Using cutting-edge technologies, intracellular components can be analyzed comprehensively to construct computer simulation models that can find numerous applications in fields such as biomedical, environmental, and agricultural science. Experimental and computational facilities are located in Tsuruoka, Yamagata prefecture, in northern Japan while the SFC campus, in the Tokyo area, hosts the bioinformatics laboratory and most undergraduate curricular activities. IAB has successfully attracted very significant funding for multiple research projects from major funding organizations including the New Energy and Industrial Technology Development Organization (NEDO) (2002-2006), for bioprocesses and cell modeling, the Ministry of Education, Culture, Sports, Science and Technology (MEXT) and its COE network, for biosimulation and systems biology (2003-2008), the Japan Science and Technology Agency (CREST, 2004-2009) for simulation and systems biology, the Ministry of Health, for cancer biology (2005), as well as from Yamagata prefecture and Tsuruoka city, 2001-2006 and 2006-2011). Over the past few years, IAB scientists have accumulated several awards including the 1st prize during the 5th Japan Biotechnology Business Competition (2005), the Minister of State for Science and Technology Policy award in recognition for industry-academia-government collaboration performance (2004), the IBM Shared University Research Award (2003), and the Nihon Kogyo Shimbunsha Award (2003) during the 17th Leading-edge Technology for Originality and Creativity. Sponsor. This study was supported by a grant from the Global COE Program entitled, Human Metabolomic Systems Biology and by a Grant-in-Aid for Scientific Research on Priority Areas Systems Genomes and on Lifesurveyor from the Ministry of Education, Culture, Sports, Science and Technology of Japan as well as research funds from the Yamagata prefectural government and the City of Tsuruoka.


https://www.addgene.org/47782/

Cite This: RRID:Addgene_47782

Species: Synthetic
Genetic insert: Codon-optimised SPATR
Vector Backbone: pTT3
Reference: PMID:24043421
Comment:


Cite this (MGI Cat# 5792408, RRID:MGI:5792408)

Source Database: MGI, catalog # 5792408
Genetic Background: involves: BALB/c * C3H/HeH * C57BL/6J
Affected Genes: Myo6
Genomic Alteration: mpc285H
Availability: Availability unknown check source stock center
Reference: PMID:27534441
Notes: abnormal inner ear vestibule morphology, deafness Allele Detail: Chemically induced (ENU)


https://web.expasy.org/cellosaurus/CVCL_8V85

Cite this: (ECACC Cat# 91060304, RRID:CVCL_8V85)
Organism: Homo sapiens
Disease:
Category: Transformed cell line DT Created: 23-02-16; Last updated: 07-09-18; Version: 3
Comment: Part of: ECACC chromosomal abnormality collection. Transformant: NCBI_TaxID; 10376; Epstein-Barr virus (EBV). DT Created: 23-02-16; Last updated: 07-09-18; Version: 3


https://www.ncbi.nlm.nih.gov/biosample/SAMN08617637

Cite this: (IIDP, Cat# 1983, RRID:SAMN08617637)
Organism: Homo sapiens
Disease: Diabetes
Category: Tissue: Pancreas
Comment: IIDP Submission for Human Islets; Provider: Luis Fernandez,University of Wisconsin, Madison, WI, 53705-2275; Age: 54.00 Years; Submission Date: 2018-02-27T14:56:04.073; Status of tissue: live


http://antibodyregistry.org/AB_417031

Cite this (R and D Systems Cat# MAB2760, RRID:AB_417031)
Comments: vendor recommendations: IgG2B Flow Cytometry; Flow Cytometry
References:
Host Organism: rat
Clonality: monoclonal antibody
Target(s): Human HM74A MAb (Clone 245106)


Cite this (GD Graphics Library, RRID:SCR_008529)

URL: http://www.boutell.com/gd/

Resource Type: Resource, software resource, data or information resource, database

GD library is an open source code library for the dynamic creation of images by programmers. GD is written in C, and wrappers are available for Perl, PHP and other languages. GD creates PNG, JPEG and GIF images, among other formats. GD is commonly used to generate charts, graphics, thumbnails, and most anything else, on the fly. While not restricted to use on the web, the most common applications of GD involve website development. gd is a graphics library. It allows your code to quickly draw images complete with lines, arcs, text, multiple colors, cut and paste from other images, and flood fills, and write out the result as a PNG or JPEG file. This is particularly useful in World Wide Web applications, where PNG and JPEG are two of the formats accepted for inline images by most browsers. gd is not a paint program. If you are looking for a paint program, you are looking in the wrong place. If you are not a programmer, you are looking in the wrong place, unless you are installing a required library in order to run an application. gd does not provide for every possible desirable graphics operation. It is not necessary or desirable for gd to become a kitchen-sink graphics package, but version 2.0 does include most frequently requested features, including both truecolor and palette images, resampling (smooth resizing of truecolor images) and so forth. What if I want to use another programming language? Not all of these tools are necessarily up to date and fully compatible with 2.0.33. PHP A variant of gd 2.x is included in PHP 4.3.0. It is also possible to patch PHP 4.2.3 for use with gd 2.0.33; see the gd home page for a link to that information. It would be a Good Idea to merge all of the things that are better in mainstream gd and all of the things that are better in PHP gd at some point in the near future. Perl gd can also be used from Perl, courtesy of Lincoln Stein''s GD.pm library, which uses gd as the basis for a set of Perl 5.x classes. Highly recommended. OCaml gd can be used from OCaml, thanks to Matt Gushee''s GD4O project. Tcl gd can be used from Tcl with John Ellson''s Gdtclft dynamically loaded extension package. Pascal Pascal enthusiasts should look into the freepascal project, a free Pascal compiler that includes gd support. REXX A gd interface for the REXX language is available. Any Language The fly interpreter performs gd operations specified in a text file. You can output the desired commands to a simple text file from whatever scripting language you prefer to use, then invoke the interpreter.


https://www.addgene.org/71388/

Cite This: RRID:Addgene_71388
UCHL5
Species: Homo sapiens
Genetic insert: hUCH37 (ISF1) M148D/F149D
Vector Backbone: pET151-D-TOPO
Reference: PMID:25702872
Comment: M148D/F149D mutations were incorporated via Quickchange mutagenesis.


Cite this (MGI Cat# 3770662, RRID:MGI:3770662)

Source Database: MGI, catalog # 3770662
Genetic Background: NOD.Cg-Prkdc Il2rg/Sz
Affected Genes: Prkdc, Il2rg
Genomic Alteration: tm1Wjl; scid
Availability: Availability unknown check source stock center
Reference: PMID:15879151, PMID:18785974
Notes: abnormal spleen B cell follicle morphology, lymph node hypoplasia, decreased mean corpuscular volume, abnormal response to transplant, decreased immature B cell number, increased cellular sensitivity to X-ray irradiation, premature death, decreased NK cell number, abnormal splenic cell ratio, decreased leukocyte cell number, abnormal response to transplant, decreased CD4-positive, alpha beta T cell number, decreased CD8-positive, alpha-beta T cell number, decreased immunoglobulin level, abnormal immune system organ morphology, abnormal thymus morphology, decreased mature B cell number, small lymph nodes Allele Detail: Spontaneous, Targeted


https://web.expasy.org/cellosaurus/CVCL_8V86

Cite this: (ECACC Cat# 91052308, RRID:CVCL_8V86)
Organism: Homo sapiens
Disease: Fragile X syndrome
Category: Transformed cell line DT Created: 23-02-16; Last updated: 07-09-18; Version: 3
Comment: Part of: ECACC chromosomal abnormality collection. Transformant: NCBI_TaxID; 10376; Epstein-Barr virus (EBV). DT Created: 23-02-16; Last updated: 07-09-18; Version: 3


https://www.ncbi.nlm.nih.gov/biosample/SAMN08482536

Cite this: (IIDP, Cat# 823, RRID:SAMN08482536)
Organism: Homo sapiens
Disease: Diabetes
Category: Tissue: Pancreas
Comment: IIDP Submission for Human Islets; Provider: Fouad Kandeel,Southern California Islet Cell Resources Center, Duarte, CA, 91010; Age: 64.00 Years; Submission Date: 2018-02-07T18:01:04.190; Status of tissue: live


http://antibodyregistry.org/AB_417373

Cite this (Millipore Cat# 05-897, RRID:AB_417373)
Comments: seller recommendations: IgM; IgM WB, IC; Immunocytochemistry; Western Blot
References:
Host Organism:
Clonality: monoclonal antibody
Target(s): HDAC9 clone LH/JC2


Cite this (York University; Ontario; Canada, RRID:SCR_006670)

URL: http://www.yorku.ca/

Resource Type: Resource, university, institution


  1. RRID Portal Resources

    Welcome to the RRID Resources search. From here you can search through a compilation of resources used by RRID and see how data is organized within our community.

  2. Navigation

    You are currently on the Community Resources tab looking through categories and sources that RRID has compiled. You can navigate through those categories from here or change to a different tab to execute your search through. Each tab gives a different perspective on data.

  3. Logging in and Registering

    If you have an account on RRID then you can log in from here to get additional features in RRID such as Collections, Saved Searches, and managing Resources.

  4. Searching

    Here is the search term that is being executed, you can type in anything you want to search for. Some tips to help searching:

    1. Use quotes around phrases you want to match exactly
    2. You can manually AND and OR terms to change how we search between words
    3. You can add "-" to terms to make sure no results return with that term in them (ex. Cerebellum -CA1)
    4. You can add "+" to terms to require they be in the data
    5. Using autocomplete specifies which branch of our semantics you with to search and can help refine your search
  5. Save Your Search

    You can save any searches you perform for quick access to later from here.

  6. Query Expansion

    We recognized your search term and included synonyms and inferred terms along side your term to help get the data you are looking for.

  7. Collections

    If you are logged into RRID you can add data records to your collections to create custom spreadsheets across multiple sources of data.

  8. Sources

    Here are the sources that were queried against in your search that you can investigate further.

  9. Categories

    Here are the categories present within RRID that you can filter your data on

  10. Subcategories

    Here are the subcategories present within this category that you can filter your data on

  11. Further Questions

    If you have any further questions please check out our FAQs Page to ask questions and see our tutorials. Click this button to view this tutorial again.

X