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What is the central question of this study? Humans with obesity have lower ATP synthesis in muscle along with lower content of the β-subunit of the ATP synthase (β-F1-ATPase), the catalytic component of the ATP synthase. Does lower synthesis rate of β-F1-ATPase in muscle contribute to these responses in humans with obesity? What is the main finding and its importance? Humans with obesity have a lower synthesis rate of β-F1 -ATPase and ATP synthase specific activity in muscle. These findings indicate that reduced production of subunits forming the ATP synthase in muscle may contribute to impaired generation of ATP in obesity.
Pubmed ID: 30362197 RIS Download
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This monoclonal targets ATPB
View all literature mentionsThis polyclonal targets GAPDH
View all literature mentionsThis polyclonal secondary targets IgG
View all literature mentionsThis monoclonal targets mouse IgGκ light chain immunoglobulins
View all literature mentionsThis monoclonal targets ATP Synthase beta
View all literature mentionsThis monoclonal targets ATPB
View all literature mentionsThis monoclonal targets ATP Synthase beta
View all literature mentionsThis polyclonal targets GAPDH
View all literature mentionsThis polyclonal secondary targets IgG
View all literature mentionsThis monoclonal targets mouse IgGκ light chain immunoglobulins
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