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Enhanced regeneration of retinal ganglion cell (RGC) axons can be achieved by modification of numerous neuronal-intrinsic factors. However, axon growth initiation and the pathfinding behavior of these axons after traumatic injury remain poorly understood outside of acute injury paradigms, despite the clinical relevance of more chronic settings. We therefore examined RGC axon regeneration following therapeutic delivery that is postponed until 2 months after optic nerve crush injury.
Pubmed ID: 28324115 RIS Download
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laboratory mouse with name C57BL/6N from MGI.
View all literature mentionsMus musculus with name B6.129X1-Baxtm1Sjk/J from IMSR.
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