MicroRNA miR-155 is implicated in modulation of the inflammatory processes in various pathological conditions. In our previous studies, we demonstrated that in vivo inhibition of miR-155 promotes functional recovery after mouse experimental stroke. In the present study, we explored if this beneficial effect is associated with miR-155 inhibition-induced alterations in post-stroke inflammatory response.
Pubmed ID: 27829437 RIS Download
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View all literature mentionsTHIS RESOURCE IS NO LONGER IN SERVICE. Documented on May 5,2022.Tool that predicts interactions between transcription factors and their regulated genes from binding motifs. Understanding vertebrate development requires unraveling the cis-regulatory architecture of gene regulation. PRISM provides accurate genome-wide computational predictions of transcription factor binding sites for the human and mouse genomes, and integrates the predictions with GREAT to provide functional biological context. Together, accurate computational binding site prediction and GREAT produce for each transcription factor: 1. putative binding sites, 2. putative target genes, 3. putative biological roles of the transcription factor, and 4. putative cis-regulatory elements through which the factor regulates each target in each functional role.
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View all literature mentionsThis polyclonal targets Mouse CD45
View all literature mentionsPublic image processing and analysis program for Macintosh.
View all literature mentionsThis polyclonal targets Mouse CD45
View all literature mentionsThis polyclonal targets Mouse MMR
View all literature mentionsThis polyclonal targets Iba1
View all literature mentionsThis polyclonal targets Mouse MMR
View all literature mentionsThis polyclonal targets Iba1
View all literature mentionsPublic image processing and analysis program for Macintosh.
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